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Download CRUCC 2.4.rar.zip & get CRUCC 2.4.rarNeuroprotective Effects of Ketamine in Chronic Alcoholism-Induced Depression.
Psychostimulants and antidepressants have been successfully used to mitigate the symptoms of depression in chronic alcoholics; however, the mechanisms underlying these effects are still not clear. The aim of the present study was to investigate the behavioral and neurochemical effects of ketamine administration in two chronic, binge-ethanol-induced depression models in male Wistar rats. In the first series of experiments, 75% ethanol was administered in a binge-ethanol-feeding protocol to induce depression in rats. The administration of ketamine (25 mg/kg, i.p.) resulted in a significant improvement in the forced swimming test in the ethanol group. In the second experiment, we also tested the effect of 20-day administration of ethanol in a subchronic binge-ethanol model. Neurochemical and behavioral studies were conducted in brain tissues after 20 days of ethanol exposure. Ketamine (25 mg/kg, i.p.) administered in both models was found to improve the depression-like behavior. The present study demonstrates the potential antidepressant effect of ketamine in two chronic alcohol-induced depression models and further highlights the relevance of this study, especially in light of its minimal side effects..
*/
int _record_method_call(JNIEnv* env, int recvr, int callId, int… argIds)
{
if (_instrumenting) {
// The native environment is instrumented, throw an exception instead.
JNIException* exception = JNU_NewException(env, “Instrumentation must be disabled”);
__android_log_print(ANDROID_LOG_WARN, DEBUG_TAG, “JNI_ERR”, “Raised by instrumentation”);
return JNI_ERR;
}
int err = 0;
err = dvmMterpInstrumentMethod(recvr, callId, argIds,
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