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Nov 21, 2014 AVIS(Akvis) Magnifier V 9.0.1188.13948 Key/Crack Free Download.]; Chen et al., [@B13]; Sando et al., [@B89]). Ultimately, clinical evidence points to oligodendrocyte dysfunction as a key component in the pathogenesis of psychiatric disorders and autism spectrum disorders (Silver and Miller, [@B94]). Deregulation of epigenetic mechanisms that are important in the regulation of OPC function might play a critical role in these neurodevelopmental disorders. In HD, altered synapse and neuronal function, and dysregulation of glutamatergic, GABAergic, dopaminergic, and noradrenergic pathways, have been observed (Ross, [@B85]). However, it is important to note that many of the HD patients that we studied in the current work displayed very subtle movement and behavioral anomalies at the time of evaluation (i.e., young adults), which may not have been apparent at that time.
Our data further provide evidence that OPC differentiation is abnormal in HD. While OPC number was increased in late symptomatic stage HD brains, the distribution of APC was not uniform, and increased OPCs with abnormal APC patterning were observed in several brain regions of both YAC128 and R6/2 mice. Moreover, in R6/2 mice, colocalization of OPCs with neurofilament-L was significantly decreased in all brain regions studied. Such changes in OPC patterning may be accompanied by loss of OPC function and the dysregulation of OPC functions (such as myelin repair) that are important in regulating myelination of axons and preventing synapse loss. Increased OPC number has been observed in some neurological disorders, including amyotrophic lateral sclerosis and Alzheimer’s disease (Kikuchi et al., [@B48]; Keseru et al., [@B47]). However, results from the current study show that altered OPC number is not specific to HD, but is likely a normal feature of neurodegenerative disease in general.
Conclusion {#s5}
==========
In summary, our data provide new evidence of several cellular and biochemical features that are abnormal in HD. We suggest that changes in OPC number and patterning contribute to the development of neuropathology in HD.
Author contributions
====================
Co-first author: Qian Chen.
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