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The formation of new blood vessels is essential for the regeneration of cartilage tissue. Stimulation of endogenous chondrocytes to secrete fibroblast growth factor-2 (FGF-2) can induce angiogenesis and chondrogenesis. In the present study, we used a culture system of chondrogenic ATDC5 cells to investigate the physiological function of FGF-2 in the regulation of chondrocyte differentiation and found that FGF-2 could inhibit the expression of type II collagen and promote the formation of capillary-like structure of endothelial cells, resulting in a significant decrease of the viability of chondrocytes. This effect may be mediated by the inactivation of vascular endothelial growth factor receptor 2 (VEGFR2). Our findings suggest that FGF-2 inhibits chondrogenesis by downregulating the expression of type II collagen and increasing the expression of VEGFR2 via downregulation of the Wnt signaling pathway. In addition, FGF-2 signaling may induce premature chondrogenesis via the phosphorylation of ERK1/2 and might eventually lead to inactivation of VEGFR2.FDP rein macht rechte Bevölkerung in Deutschland

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Use a for loop:
for i = 1:4
if i==2
plot(time,array_plus(‘line_plus’,array_plus(‘square_plus’,array_plus(‘triangle_plus’,… ‘wave_plus’),… ‘wave_plus’),… ‘square_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘wave_plus’));
elseif i==3
plot(time,array_plus(‘square_plus’,array_plus(‘line_plus’,array_plus(‘triangle_plus’,…’square_plus’),… ‘triangle_plus’),… ‘line_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,… ‘triangle_plus’,… ‘line_plus’,… ‘square_plus’,…
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**To the Editor:** Epidemic myalgia and myositis is caused by *Campylobacter jejuni* that causes “new cases of Guillain-Barré syndrome in a country previously considered free of these conditions” ([@R1]). Since first described in 1935 by Halliday and Ewing, this syndrome has been reported in children and adults in several countries ([@R2]–[@R7]). Symptomatic *C. jejuni* enteritis usually occurs 2–6 weeks after onset of neurologic symptoms. However, severe acute diarrheal illness is often the initial manifestation of *C. jejuni* infection. Frequently, diarrhea will have been present in a person with acute myositis for up to 10 weeks before it is recognized ([@R8],[@R9]).

We report a case of epidemic myalgia and myositis caused by *C. jejuni* infection in a 15-year-old boy from Saudi Arabia. The boy had no recent travel history and no contact with any known patient with this illness. He presented to the emergency room of a regional hospital with a 1-day history of progressive left thigh and leg weakness and pain. The symptoms developed while he was playing basketball, and he was unable to go back to school. He was admitted to the hospital and prescribed parenteral antimicrobial drugs. Within 3 days after admission, he developed diarrhea, and the fever and abdominal pain were no longer apparent. After the first episode of diarrhea, he became intermittently unable to walk. Acute viral illness and trauma were excluded.

Muscle biopsy performed on day 7 of admission showed moderate muscle fiber necrosis, myophagocytosis with neutrophil infiltration, and increased mitochondrial volume density. Prednisone (60 mg)

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